-->The multivariate analysis found that Chest x-ray findings of ARDS vs. no. finding was significant between both groups (OR 2.03 (1.07-3.86), P =0.03?). Is this difference explained by possible decreased capacity of lung to convert angiotensin ATI to ATII, would that affect dosing requirements & expectation of response in this subgroup?
-->Multivariate analysis found that patients with prior exposure to ARBs vs. no exposure had significantly different responses, (OR 0.24 (0.07-0.79) P=0.02). Would prior exposure to ACEi, result in upregulation of ATII receptors – and thus mean these patients should be dosed more cautiously?
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