Question special

The study found that -- not surprisingly -- the prevalence of tachypnea, tachycardia, and hypotension was higher among patients with PE than among patients without PE. But it also found that about 25% of patients with PE had NO clinical manifestations of the diagnosis, which is a worrying statistic for clinicians.

How should we think about this subset of patients with PE? Do we know if these patients were the ones who had lower thrombotic burden on imaging -- and perhaps for whom PE wasn't the factor driving their syncope?